G. IPF and scleroderma), but the pathophysiology of those versions, relying on immediate administration of drug to tissue, differs greatly from clinical bleomycin‐induced lung fibrosis. Alternatively, systemic administration of a pro‐fibrotic drug like bleomycin ought to more closely mimic the inflammatory and fibrotic procedures witnessed in humans inside the context https://quincyh554ufp7.bloggactivo.com/profile